By J. Larry Jameson, Joseph Loscalzo
Nephrology and Acid-Base problems – as purely Harrison’s can hide it
Featuring an outstanding compilation of chapters with regards to kidney functionality that seem in Harrison’s ideas of inner drugs, Eighteenth Edition, this concise, full-color medical spouse provides the most recent wisdom within the box subsidized by means of the medical rigor and authority that experience outlined Harrison’s. you can find content material from popular editors and individuals in a carry-anywhere presentation that's perfect for the school room, health center, ward, or exam/certification preparation.
- Sections that replicate the scope of nephrology: creation to the Renal procedure; changes of Renal functionality and Electrolytes; Acute Kidney harm and persistent Renal Failure; Glomerular and Tubular problems; Renal Vascular illness; Urinary Tract Infections and Obstruction; and melanoma of the Kidney and Urinary Tract
- Complete assurance of a large spectrum of subject matters, together with acid-base and electrolyte problems, vascular damage to the kidney, and particular illnesses of the kidney
- Integration of pathophysiology with scientific administration
- 41 high-yield questions and solutions drawn from Harrison’s ideas of inner medication Self-Assessment and Board evaluate, 18e
- Content updates and new advancements because the booklet of Harrison’s ideas of inner medication, 18e
- 22 chapters written via physicians who're well-known specialists within the box of nephrology and acid-base issues
- Helpful appendix of laboratory values of scientific importance
Read Online or Download Harrison's Nephrology and Acid-Base Disorders, 2e PDF
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Additional resources for Harrison's Nephrology and Acid-Base Disorders, 2e
The finding of eosinophils in the urine is Intrinsic 27 EVALUATION OF HEMATURIA HEMATURIA Proteinuria (>500 mg/24 h), Dysmorphic RBCs or RBC casts Pyuria, WBC casts Urine culture Urine eosinophils Hemoglobin electrophoresis Urine cytology UA of family members 24-h urinary calcium/uric acid As indicated: retrograde pyelography or arteriogram, or cyst aspiration Cystoscopy Urogenital biopsy and evaluation Renal CT scan Renal biopsy of mass/lesion Renal biopsy Follow periodic urinalysis Figure 3-2 Approach to the patient with hematuria.
The etiologies of prerenal azotemia include any cause of decreased circulating blood volume (gastrointestinal hemorrhage, burns, diarrhea, diuretics), volume sequestration (pancreatitis, peritonitis, rhabdomyolysis), or decreased effective arterial volume (cardiogenic shock, sepsis). Renal perfusion also can be affected by reductions in cardiac output from peripheral vasodilation (sepsis, drugs) or profound renal vasoconstriction [severe heart failure, hepatorenal syndrome, drugs such as nonsteroidal anti-inflammatory drugs (NSAIDs)].
Figure 4-3 Collapsing glomerulopathy. There is segmental collapse of the glomerular capillary loops and overlying podocyte hyperplasia. This lesion may be idiopathic or associated with HIV infection and has a particularly poor prognosis. ) Figure 4-2 Focal segmental glomerulosclerosis (FSGS). There is a well-defined segmental increase in matrix and obliteration of capillary loops, the sine qua non of segmental sclerosis not otherwise specified (nos) type. ) 32 33 A B Figure 4-6 Postinfectious (poststreptococcal) glomerulonephritis.