By Nigel Lane
Best of 5 MCQs for the intense drugs SCE is a brand new revision source designed particularly for this high-stakes examination. Containing over 350 better of 5 a number of selection questions, this devoted consultant can assist applicants to organize effectively.
The content material mirrors the SCE in Acute drugs Blueprint to make sure applicants are absolutely ready for all of the issues that can look within the examination. themes variety from how you can deal with acute difficulties in cardiology or neurology to handling acute stipulations reminiscent of poisoning. All solutions have complete reasons and extra analyzing to make sure top of the range self-assessment and quickly acceptance of parts that require additional study.
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Extra resources for Best of five mcqs for the acute medicine sce
As the core temperature nears normal levels shivering may begin and require treatment with sedation and analgesia. Hypothermia per se is associated with complications such as pancreatitis, immune suppression and infection, and coagulopathy. Do not forget to look for an underlying cause which could be a medical or surgical illness, or trauma- or drug-related. Brown D, Brugger H, Boyd J, Paal P. Accidental hypothermia. New England Journal of Medicine 2012; 367: 1930–1938. 16. Anaphylaxis–adrenaline Anaphylaxis may not present on the first administration of the offending drug.
PH B. Prothrombin time C. Serum alanine aminotransferase D. Serum bilirubin E. Serum creatinine 20. A 60-year-old man with alcoholic liver disease was admitted to the acute medical unit with ascites. He asks you about liver transplantation, what should you tell him? Abstinence from alcohol for six months is mandatory A B. Alcoholic cardiomyopathy is a major determinant of outcome C. Most patients start drinking again after transplantation D. One-year survival is around 65% E. Patients more than 60 years old are not eligible 21.
In the absence of peripheral or pulmonary oedema (the latter very unlikely with normal saturations and respiratory rate) there is no evidence of fluid overload either. Finally, the likely renal injury is contrast medium-related and therefore unlikely to respond to fluid loading after the event. Second, the central venous pressure (CVP) is not a measure of intravascular volume. It is a pressure measurement that is determined by the relationship between cardiac performance and venous return. Poor function and low return cannot be distinguished from good function and normal return or hyperdynamic function and increased return on the basis of a static reading.