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By Jeff Coulter

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Extra resources for Approaches to Insanity: A Philosophical and Sociological Study

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The most familiar of the causal accounts is that which reduces the process of human socialisation to the level of animal conditioning; a simple over-extrapolation of Pavlovian reflexology to the normative order of human life experience and interaction. Conditioning theory has never flourished in psychopathology, apart from the early work of Meyer 3 and the occasional use of behaviouristic epistemology for rationalising certain kmds of therapeutic regimes. 4 Eysenck's contributions have remained largely restricted to the 'neuroses' ,5 and need not concern us here.

See M. Siegel et al, op. , and R. K. McDonald's paper, 'Plasma Ceruloplasmin and Ascorbic Acid Levels in Schizophrenia' (Presented to the 1957 Annual Meeting of the American Psychiatric Association in Chicago and cited inS. S. Kety, op. ) McDonald found that where high ceruloplasmin levels were characteristic of a sample of diagnosed schizophrenics it could be completely accounted for by a dietary deficiency of ascorbic acid. 44. See A. A. Boulton, op. cit. pp. 25-26 45. F. 0. A. Archives of Neurology and Psychiatry, Vol.

58. C. Rosenbaum; paper in Journal of Nervous and Mental Disorders vol. 146 (1968); cited in Boulton 59. A. R. Kaplan in Nature vol. 210 (1966) Schizophrenia as a Disease 33 60. R. J. Baldessarini & S. H. Snyder in Nature vol. 206 (1965) 61. F. J. Kallman 'The Genetic Theory of Schizophrenia: An Analysis of 691 Schizophrenic Twin Index Families' American Journal of Psychiatry vol. 103 (1946); F. J. Kallman Heredity in Health and Mental Disorder New York, Norton (1953) 62. D. D. Jackson 'A Critique of the Literature on the Genetics of Schizophrenia' in D.

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